{"id":1142,"date":"2025-03-10T13:29:45","date_gmt":"2025-03-10T13:29:45","guid":{"rendered":"https:\/\/urolistic.com\/?p=1142"},"modified":"2025-05-27T08:07:59","modified_gmt":"2025-05-27T08:07:59","slug":"el-papel-del-intestino-y-su-microbiota-eje-intestino-cerebro","status":"publish","type":"post","link":"https:\/\/urolistic.com\/fr\/blog\/2025\/03\/10\/el-papel-del-intestino-y-su-microbiota-eje-intestino-cerebro\/","title":{"rendered":"Le r\u00f4le de l'intestin et de son microbiote. L'axe intestin-cerveau"},"content":{"rendered":"<div data-elementor-type=\"wp-post\" data-elementor-id=\"1142\" class=\"elementor elementor-1142\" data-elementor-post-type=\"post\">\n\t\t\t\t<div class=\"elementor-element elementor-element-75f0c6b2 e-flex e-con-boxed e-con e-parent\" data-id=\"75f0c6b2\" data-element_type=\"container\" data-e-type=\"container\">\n\t\t\t\t\t<div class=\"e-con-inner\">\n\t\t\t\t<div class=\"elementor-element elementor-element-7b7b83b elementor-widget elementor-widget-text-editor\" data-id=\"7b7b83b\" data-element_type=\"widget\" data-e-type=\"widget\" data-widget_type=\"text-editor.default\">\n\t\t\t\t<div class=\"elementor-widget-container\">\n\t\t\t\t\t\t\t\t\t\n<p>L'\u00e9tude du microbiote intestinal est un domaine en plein essor depuis quelques ann\u00e9es. En plus de favoriser une bonne sant\u00e9 digestive (meilleure digestion, meilleur transit intestinal, diminution du risque de cancer du c\u00f4lon, diminution des sympt\u00f4mes gastro-intestinaux tels que les br\u00fblures d'estomac, les ballonnements, etc.), la pr\u00e9sence d'un microbiote intestinal \"sain\" a \u00e9t\u00e9 associ\u00e9e \u00e0 de multiples bienfaits pour la sant\u00e9. Aujourd'hui, on parle souvent de l'axe microbiote-intestin-cerveau, et l'on consid\u00e8re que le fait d'avoir un bon microbiote intestinal nous permet d'avoir de meilleures fonctions neurologiques, psychologiques, immunologiques ou hormonales, entre autres.<\/p>\n\n<p>\u00a0\u00a0\u00a0\u00a0\u00a0Dans l'intestin, il existe un vaste r\u00e9seau de terminaisons nerveuses provenant du syst\u00e8me nerveux autonome sympathique et parasympathique, en particulier le nerf vague et les nerfs de la moelle \u00e9pini\u00e8re, ainsi qu'un r\u00e9seau neuronal intrins\u00e8que, appel\u00e9 syst\u00e8me nerveux ent\u00e9rique. Ces r\u00e9seaux sont interconnect\u00e9s. Par la lib\u00e9ration de nombreuses substances chimiques (neurotransmetteurs, acides gras \u00e0 cha\u00eene courte, peptides, hormones, cytokines, etc.), le nerf vague permet une communication constante entre le cerveau et l'intestin. La communication est bidirectionnelle : le nerf vague lib\u00e8re des substances dans l'intestin s'il s'agit d'une information eff\u00e9rente et l'intestin transmet d'autres substances chimiques au nerf vague pour que l'information parvienne au cerveau, s'il s'agit d'un message aff\u00e9rent. Ces substances chimiques peuvent \u00eatre produites \u00e0 la fois par les terminaisons nerveuses, les cellules intestinales ou immunitaires de la paroi intestinale et, dans une large mesure, par le microbiote. Le microbiote joue donc un r\u00f4le crucial dans la communication entre l'intestin et le cerveau. Je tiens \u00e0 pr\u00e9ciser que le nerf vague, s'il est la principale voie de communication entre l'intestin et le cerveau, n'est en aucun cas la seule. Une autre voie de communication importante est la voie sanguine (les hormones, cytokines ou autres substances chimiques produites dans le cerveau se rendent dans l'intestin par le biais du sang, et vice versa). Je n'irai pas plus loin dans le monde passionnant de l'axe intestin-cerveau, qui pourrait faire l'objet de plusieurs livres, mais, \u00e0 titre d'exemple, je pourrais dire qu'il est plus que prouv\u00e9 que les personnes souffrant de maladies psychiatriques telles que la d\u00e9pression ou l'anxi\u00e9t\u00e9, de maladies neurod\u00e9g\u00e9n\u00e9ratives telles que la maladie d'Alzheimer ou de Parkinson ou de troubles neurod\u00e9veloppementaux (troubles du spectre autistique par exemple) pr\u00e9sentent souvent une alt\u00e9ration profonde de leur microbiote intestinal, appel\u00e9e \"dysbiose\".\u00a0<\/p>\n\n<p>\u00a0\u00a0\u00a0\u00a0L'intestin abrite environ 80% des cellules immunitaires de notre corps, car il s'agit du principal \"poste de douane\" de notre organisme, o\u00f9 ont lieu les plus grands \u00e9changes avec le monde ext\u00e9rieur. L'interaction entre le microbiote et les cellules immunitaires intestinales d\u00e8s les premiers jours de la vie est essentielle pour que l'immunit\u00e9 d'une personne se d\u00e9veloppe normalement. Le syst\u00e8me immunitaire apprend ainsi \u00e0 tol\u00e9rer les micro-organismes qui font partie de notre microbiote et qui nous aident dans de multiples fonctions de notre corps, et \u00e0 attaquer les micro-organismes qui sont dangereux pour notre sant\u00e9.\u00a0<\/p>\n\n<p>\u00a0\u00a0\u00a0\u00a0\u00a0Le microbiote intestinal est \u00e9galement notre grand alli\u00e9 pour dig\u00e9rer et assimiler correctement de nombreux aliments que nous consommons. Une partie de la digestion de certaines mol\u00e9cules de notre alimentation, notamment les glucides et certaines prot\u00e9ines, est r\u00e9alis\u00e9e par certains micro-organismes. Plus important encore, ces micro-organismes fabriquent ce que l'on appelle des acides gras \u00e0 cha\u00eene courte (AGCC) (butyrate, propionate et ac\u00e9tate), dont on pense qu'ils ont de nombreux effets sur notre organisme. En effet, on pense aujourd'hui que les AGCC sont capables de r\u00e9guler entre 5% et 20% de nos g\u00e8nes, agissant ainsi sur notre m\u00e9tabolisme ainsi que sur la diff\u00e9renciation et la prolif\u00e9ration des cellules. Ils r\u00e9gulent \u00e9galement la r\u00e9ponse immunitaire de l'intestin, comme mentionn\u00e9 plus haut, la production d'hormones par les cellules de la paroi intestinale et la motilit\u00e9 intestinale, en favorisant le transit. Ils constituent \u00e9galement une importante source d'\u00e9nergie, en particulier pour les cellules de la paroi intestinale. Les micro-organismes de notre microbiote fabriquent \u00e9galement des neurotransmetteurs, comme je l'ai d\u00e9j\u00e0 mentionn\u00e9, et des vitamines (comme la vitamine K ou les vitamines du groupe B, principalement la B12). De m\u00eame, les \"bons\" micro-organismes du microbiote produisent des substances, comme le peroxyde d'hydrog\u00e8ne ou la bact\u00e9riocine, qui tuent ou inhibent la croissance d'autres micro-organismes \"mauvais\" ou \"moins bons\". Vous pouvez donc vous faire une id\u00e9e de l'importance d'avoir un microbiote sain, sans dysbiose.<\/p>\n\n<h3 id=\"h-relacion-microbiota-intestinal-y-sistema-urogenital\" class=\"wp-block-heading\">Relation entre le microbiote intestinal et le syst\u00e8me urog\u00e9nital<\/h3>\n\n<p>La pr\u00e9sence de certains micro-organismes intestinaux peut d\u00e9clencher diff\u00e9rentes r\u00e9actions immunologiques ou neuro-hormonales qui affectent l'\u00e9tat neuro-immuno-hormonal de notre organisme, et plus particuli\u00e8rement celui de notre syst\u00e8me urinaire. Un \u00e9tat d'inflammation chronique de bas grade provoqu\u00e9 par la consommation d'aliments pro-inflammatoires (c\u00e9r\u00e9ales, produits laitiers de vache, produits contenant des substances toxiques, etc.) ou par la pr\u00e9sence de certains micro-organismes pro-inflammatoires peut g\u00e9n\u00e9rer une r\u00e9ponse immunitaire syst\u00e9mique alt\u00e9r\u00e9e, par exc\u00e8s ou par d\u00e9faut, une alt\u00e9ration du taux de certaines hormones et neurotransmetteurs comme le cortisol ou la s\u00e9rotonine, et une propension aux infections urinaires ou \u00e0 d'autres probl\u00e8mes urologiques. De m\u00eame, une production insuffisante par le microbiote de certains neurotransmetteurs \"relaxants\" comme le GABA pourrait th\u00e9oriquement conduire \u00e0 une hypertonie musculaire et \u00e0 un d\u00e9ficit cons\u00e9cutif de relaxation sphinct\u00e9rienne, favorisant l'apparition ou l'aggravation de mictions non coordonn\u00e9es ou de constipation.\u00a0<\/p>\n\n<p>\u00a0\u00a0\u00a0\u00a0\u00a0La relation entre l'alimentation, le microbiote intestinal et le syst\u00e8me urinaire est tr\u00e8s complexe et nous sommes encore loin de bien la comprendre. Depuis quelques ann\u00e9es, on soup\u00e7onne qu'il existe un \u00e9change constant d'informations entre le microbiote intestinal, urinaire et vaginal (dans le cas des femmes) ou prostatique (dans le cas des hommes). Cette connexion multidirectionnelle est plus complexe qu'on ne le pensait, dans la mesure o\u00f9 la composition d'un microbiote peut influencer les autres, m\u00eame sans transfert \"direct\" de micro-organismes. De nombreuses recherches sont encore n\u00e9cessaires pour bien comprendre ces m\u00e9canismes.<\/p>\n\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size wp-elements-203083d460e1f51a89d80dbaccad1aad\"><em>Bibliographie :<\/em><\/p>\n\t\t\t\t\t\t\t\t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t<div class=\"elementor-element elementor-element-0d4f0e4 biblio elementor-widget elementor-widget-text-editor\" data-id=\"0d4f0e4\" data-element_type=\"widget\" data-e-type=\"widget\" data-widget_type=\"text-editor.default\" data-no-translation=\"\" data-no-auto-translation=\"\">\n\t\t\t\t<div class=\"elementor-widget-container\">\n\t\t\t\t\t\t\t\t\t<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Arponen S (2021). Es la microbiota, idiota. Alienta.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Maynard CL, Elson CO, Hatton RD, Weaver CT. Reciprocal interactions of the intestinal microbiota and immune system. Nature. 2012 Sep 13;489(7415):231-41.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Cryan JF, O\u2019Riordan KJ, Cowan CSM, Sandhu KV, Bastiaanssen TFS, et al. The Microbiota-Gut-Brain Axis. Physiol Rev. 2019 Oct 1;99(4):1877-2013.\u00a0<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Takiishi T, Fenero CIM, C\u00e2mara NOS. Intestinal barrier and gut microbiota: Shaping our immune responses throughout life. Tissue Barriers. 2017 Oct 2;5(4)<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Martin-Gallausiaux C, Marinelli L, Blottiere HH, Larraufie P, Lapaque N. Short Chain Fatty Acids \u2013 mechanisms and functional importance in the gut.\u00a0Proceedings of the Nutrition Society, 2021, 80 (1), pp. 37-49.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Helander HF, F\u00e4ndriks L. Surface area of the digestive tract \u2013 revisited. Scand J Gastroenterol. 2014 Jun;49(6):681-9.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Maynard CL, Elson CO, Hatton RD, Weaver CT. Reciprocal interactions of the intestinal microbiota and immune system. Nature. 2012 Sep 13;489(7415):231-41.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Cryan JF, O\u2019Riordan KJ, Cowan CSM, Sandhu KV, Bastiaanssen TFS, et al. The Microbiota-Gut-Brain Axis. Physiol Rev. 2019 Oct 1;99(4):1877-2013.\u00a0<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Takiishi T, Fenero CIM, C\u00e2mara NOS. Intestinal barrier and gut microbiota: Shaping our immune responses throughout life. Tissue Barriers. 2017 Oct 2;5(4).<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Mitchell NM, Johnson JR, Johnston B, Curtiss R 3rd, Mellata M. Zoonotic potential of Escherichia coli isolates from retail chicken meat products and eggs. Appl Environ Microbiol. 2015 Feb;81(3):1177-87.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Buberg ML, Mo SS, Sekse C, Sunde M, Wasteson Y, Wits\u00f8 IL. Population structure and uropathogenic potential of extended-spectrum cephalosporin-resistant Escherichia coli from retail chicken meat. BMC Microbiol. 2021 Mar 29;21(1):94.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Magruder M, Sholi AN, Gong C, et al. Gut uropathogen abundance is a risk factor for development of bacteriuria and urinary tract infection.\u00a0Nat Commun. 2019;10(1):5521.\u00a0<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Magruder M, Edusei E, Zhang L, et al. Gut commensal microbiota and decreased risk for\u00a0Enterobacteriaceae\u00a0bacteriuria and urinary tract infection.\u00a0Gut Microbes. 2020;12(1):1805281.\u00a0<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Spaulding CN, Klein RD, Ruer S, et al. Selective depletion of uropathogenic E. coli from the gut by a FimH antagonist.\u00a0Nature. 2017;546(7659):528-532.\u00a0<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Jones-Freeman B, Chonwerawong M, Marcelino VR, Deshpande AV, Forster SC, Starkey MR. The microbiome and host mucosal interactions in urinary tract diseases. Mucosal Immunol. 2021 Jul;14(4):779-792.\u00a0<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Mitchell NM, Johnson JR, Johnston B, Curtiss R 3rd, Mellata M. Zoonotic potential of Escherichia coli isolates from retail chicken meat products and eggs. Appl Environ Microbiol. 2015 Feb;81(3):1177-87.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Buberg ML, Mo SS, Sekse C, Sunde M, Wasteson Y, Wits\u00f8 IL. Population structure and uropathogenic potential of extended-spectrum cephalosporin-resistant Escherichia coli from retail chicken meat. BMC Microbiol. 2021 Mar 29;21(1):94.<\/p>\t\t\t\t\t\t\t\t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t<\/div>","protected":false},"excerpt":{"rendered":"<p>El estudio de la microbiota intestinal es un campo en auge desde hace algunos a\u00f1os. Adem\u00e1s de ayudar a una buena salud digestiva (mejores digestiones, mejor<span class=\"excerpt-hellip\"> [\u2026]<\/span><\/p>\n","protected":false},"author":1,"featured_media":1176,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[14],"tags":[],"class_list":["post-1142","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-microbiota"],"yoast_head":"<!-- This site is optimized with the Yoast SEO Premium plugin v24.7 (Yoast SEO v27.4) - https:\/\/yoast.com\/product\/yoast-seo-premium-wordpress\/ -->\n<title>El papel del intestino y su microbiota. Eje intestino-cerebro | Urolistic<\/title>\n<meta name=\"description\" content=\"Redescubre el bienestar con Urolistic, el proyecto de la Dra. 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