{"id":1459,"date":"2025-05-26T09:42:21","date_gmt":"2025-05-26T09:42:21","guid":{"rendered":"https:\/\/urolistic.com\/?p=1459"},"modified":"2025-05-27T08:05:22","modified_gmt":"2025-05-27T08:05:22","slug":"comprender-la-estructura-del-intestino","status":"publish","type":"post","link":"https:\/\/urolistic.com\/fr\/blog\/2025\/05\/26\/comprender-la-estructura-del-intestino\/","title":{"rendered":"Comprendre la structure de l'intestin"},"content":{"rendered":"<div data-elementor-type=\"wp-post\" data-elementor-id=\"1459\" class=\"elementor elementor-1459\" data-elementor-post-type=\"post\">\n\t\t\t\t<div class=\"elementor-element elementor-element-5401b86d e-flex e-con-boxed e-con e-parent\" data-id=\"5401b86d\" data-element_type=\"container\" data-e-type=\"container\">\n\t\t\t\t\t<div class=\"e-con-inner\">\n\t\t\t\t<div class=\"elementor-element elementor-element-736d7f0 elementor-widget elementor-widget-text-editor\" data-id=\"736d7f0\" data-element_type=\"widget\" data-e-type=\"widget\" data-widget_type=\"text-editor.default\">\n\t\t\t\t<div class=\"elementor-widget-container\">\n\t\t\t\t\t\t\t\t\t<p>La paroi intestinale est constitu\u00e9e de plusieurs couches de tissus et de cellules sp\u00e9cialis\u00e9s dont la disposition sous forme de petits plis appel\u00e9s villosit\u00e9s intestinales lui permet de faciliter la digestion et l'absorption des nutriments. De l'int\u00e9rieur vers l'ext\u00e9rieur, on trouve la muqueuse, la sous-muqueuse, la couche musculaire et la s\u00e9reuse, la couche la plus externe. L'\u00e9pith\u00e9lium de la muqueuse intestinale est constitu\u00e9 d'une seule couche de cellules. Il est tapiss\u00e9 de mucus, une substance qui lui conf\u00e8re une protection et lui permet d'h\u00e9berger de nombreuses bact\u00e9ries du microbiote intestinal. Les cellules les plus nombreuses sont les ent\u00e9rocytes, cellules hautes et \u00e9troites, qui sont responsables de l'absorption des nutriments. La surface des ent\u00e9rocytes en contact avec la lumi\u00e8re intestinale et le mucus n'est pas lisse, mais forme de petites protub\u00e9rances appel\u00e9es microvillosit\u00e9s, qui augmentent consid\u00e9rablement la surface d'absorption des nutriments. C'est ce qu'on appelle aussi la \"bordure en brosse\". En outre, ces microvillosit\u00e9s contiennent certaines enzymes digestives telles que la lactase (qui nous permet de dig\u00e9rer le lactose du lait), la maltase (qui dig\u00e8re le maltose), la sucrase (qui dig\u00e8re le saccharose) ou l'aminopeptidase (qui dig\u00e8re les petites prot\u00e9ines appel\u00e9es di- ou tripeptides et les acides amin\u00e9s). Parmi les ent\u00e9rocytes, on trouve des cellules caliciformes, responsables de la production et de la s\u00e9cr\u00e9tion de mucus.  Il y a \u00e9galement des cellules endocrines, qui lib\u00e8rent des hormones telles que la s\u00e9cr\u00e9tine et la chol\u00e9cystokinine, qui contr\u00f4lent la s\u00e9cr\u00e9tion d'enzymes digestives par le pancr\u00e9as et la v\u00e9sicule biliaire. Dans les cryptes intestinales, la partie la plus profonde des villosit\u00e9s, se trouvent des cellules souches qui se divisent et se diff\u00e9rencient au fur et \u00e0 mesure que les cellules de surface meurent. Ces cellules sont responsables du renouvellement et de la r\u00e9g\u00e9n\u00e9ration de l'\u00e9pith\u00e9lium intestinal. Sous la muqueuse se trouve la sous-muqueuse, constitu\u00e9e de tissu conjonctif et de capillaires sanguins. Elle est responsable de l'apport de nutriments et d'oxyg\u00e8ne aux cellules de l'intestin, ainsi que de la collecte et du transport des produits de la digestion par la circulation sanguine. La couche suivante est la couche musculaire, qui se divise en deux sous-couches : la couche circulaire interne et la couche longitudinale externe. Ces couches sont constitu\u00e9es de cellules musculaires lisses, dont les mouvements sont r\u00e9gis par les nerfs du syst\u00e8me nerveux ent\u00e9rique (li\u00e9s au syst\u00e8me nerveux autonome sympathique et parasympathique) et sont responsables des mouvements de l'intestin. Ces mouvements, tels que le p\u00e9ristaltisme, la segmentation, le complexe moteur migratoire, la motilit\u00e9 colique ou les r\u00e9flexes, nous permettent de dig\u00e9rer correctement, de transporter les aliments le long du tractus gastro-intestinal et de les \u00e9liminer dans les f\u00e8ces. La couche la plus externe de la paroi intestinale est la s\u00e9reuse, compos\u00e9e de tissu conjonctif et de cellules \u00e9pith\u00e9liales. Elle contient des vaisseaux sanguins et est responsable de la protection et du soutien de l'intestin.<\/p>\n<p>\u00a0L'intestin poss\u00e8de son propre syst\u00e8me nerveux, appel\u00e9 \"syst\u00e8me nerveux ent\u00e9rique\". Il est compos\u00e9 d'environ 80 \u00e0 100 millions de neurones, soit autant que la moelle \u00e9pini\u00e8re. Il a la capacit\u00e9 de fonctionner de mani\u00e8re ind\u00e9pendante, mais il est \u00e9galement reli\u00e9 au syst\u00e8me nerveux central par le biais du syst\u00e8me nerveux autonome (sympathique et parasympathique). Il se compose de deux \u00e9l\u00e9ments principaux : le plexus sous-muqueux de Meissner, situ\u00e9 sous la sous-muqueuse, et le plexus myent\u00e9rique d'Auerbach, situ\u00e9 entre les couches musculaires circulaire et longitudinale. Le plexus de Meissner est le plus d\u00e9velopp\u00e9 dans l'intestin gr\u00eale et le c\u00f4lon.<\/p>\n<p>\u00a0Il s'occupe principalement de la r\u00e9gulation de la digestion et de l'absorption au niveau de la muqueuse et des vaisseaux sanguins, en fonction de la stimulation produite par les nutriments. Le plexus d'Auerbach coordonne l'activit\u00e9 des couches musculaires pour permettre le transit intestinal dont j'ai parl\u00e9 plus haut.<\/p>\n<p>\u00a0\u00a0\u00a0\u00a0\u00a0 \u00a0&#8211; \u00a0\u00a0\u00a0\u00a0\u00a0 <u>\u00a0Intestin poreux ou \"Leaky - gut\" (fuites intestinales)<\/u><\/p>\n<p>\u00a0La paroi intestinale a une structure complexe. Elle est tapiss\u00e9e d'une seule couche de cellules rectangulaires appel\u00e9es ent\u00e9rocytes.  Ces cellules sont li\u00e9es entre elles par des prot\u00e9ines appel\u00e9es \"jonctions serr\u00e9es\" sur leurs parois lat\u00e9rales et sont imperm\u00e9ables au passage des substances, sauf si les ent\u00e9rocytes \"autorisent\" leur ouverture. Ainsi, la plupart des substances que nous absorbons passent \u00e0 travers ces cellules pour p\u00e9n\u00e9trer dans notre corps, plut\u00f4t qu'entre elles, ce qui permet de mieux contr\u00f4ler ce qui passe et ce qui ne passe pas.  Ces cellules poss\u00e8dent \u00e9galement une \"bordure en brosse\" au sommet (la partie qui fait face \u00e0 la lumi\u00e8re intestinale), constitu\u00e9e de petits poils appel\u00e9s microvillosit\u00e9s qui leur permettent d'absorber de nombreuses substances de mani\u00e8re tr\u00e8s efficace. En outre, \u00e0 la surface des microvillosit\u00e9s se trouvent certaines enzymes qui aident \u00e0 terminer la digestion des aliments, dont la digestion a commenc\u00e9 dans la lumi\u00e8re de l'estomac ou de l'intestin gr\u00e2ce aux sucs gastriques ou pancr\u00e9atiques, qui sont des liquides charg\u00e9s d'enzymes digestives qui sont lib\u00e9r\u00e9s lorsque nous mangeons. C'est le cas par exemple du lactose, le sucre contenu dans les produits laitiers, qui est dig\u00e9r\u00e9 au niveau des microvillosit\u00e9s intestinales gr\u00e2ce \u00e0 une enzyme appel\u00e9e lactase, et dont le d\u00e9ficit peut conduire \u00e0 la fameuse intol\u00e9rance au lactose.  En cas d'inflammation chronique de la paroi intestinale, les ent\u00e9rocytes peuvent mourir ou perdre leurs microvillosit\u00e9s, ce qui peut entra\u00eener une malabsorption, une mauvaise digestion, voire une intol\u00e9rance (par exemple, l'intol\u00e9rance au lactose peut \u00eatre g\u00e9n\u00e9tique si le g\u00e8ne codant pour la lactase est manquant, ou acquise suite \u00e0 une inflammation intestinale chronique excessive). De m\u00eame, des \"pores\" peuvent s'ouvrir entre les cellules en raison de la perte ou du mauvais fonctionnement des jonctions serr\u00e9es, de sorte que l'imperm\u00e9abilit\u00e9 de ces jonctions est perdue. Il existe \u00e9galement des mol\u00e9cules telles que la gliadine du gluten (la partie non soluble dans l'eau du gluten) qui ont la capacit\u00e9 de se lier directement aux jonctions serr\u00e9es et de les ouvrir. Ainsi, m\u00eame en l'absence d'inflammation, ces aliments peuvent produire une porosit\u00e9 intestinale \"per se\". La porosit\u00e9 intestinale permet \u00e0 des substances qui devraient normalement rester dans la lumi\u00e8re intestinale et ne pas p\u00e9n\u00e9trer dans notre organisme, telles que des toxines ou des morceaux de la paroi de certaines bact\u00e9ries intestinales appel\u00e9s lipopolysaccharides, de passer la barri\u00e8re intestinale sans aucun contr\u00f4le de la part des ent\u00e9rocytes. Ainsi, ces substances provoquent directement une r\u00e9action inflammatoire locale ou passent m\u00eame dans la circulation sanguine en provoquant une inflammation \u00e0 distance. Par exemple, les lipopolysaccharides peuvent traverser la barri\u00e8re h\u00e9mato-enc\u00e9phalique, atteindre le cerveau et provoquer une neuroinflammation (inflammation du syst\u00e8me nerveux central). Ce serait l'un des m\u00e9canismes favorisant l'apparition de troubles neurod\u00e9veloppementaux chez l'enfant, comme l'autisme par exemple, ou de maladies neurod\u00e9g\u00e9n\u00e9ratives comme la maladie de Parkinson ou la maladie d'Alzheimer chez l'adulte.<\/p>\n<p><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size wp-elements-67a0e480a77206a1341e4e555813485e wp-block-paragraph\"><em>Bibliographie :<\/em><\/p>\n<p><\/p>\n<p><!-- \/wp:paragraph --><\/p>\t\t\t\t\t\t\t\t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t<div class=\"elementor-element elementor-element-60ea116 biblio elementor-widget elementor-widget-text-editor\" data-id=\"60ea116\" data-element_type=\"widget\" data-e-type=\"widget\" data-widget_type=\"text-editor.default\" data-no-translation=\"\" data-no-auto-translation=\"\">\n\t\t\t\t<div class=\"elementor-widget-container\">\n\t\t\t\t\t\t\t\t\t<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Arponen S (2021).\u00a0<em>Es la microbiota, idiota<\/em>. Alienta.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Godaly G, Ambite I, Svanborg C. Innate immunity and genetic determinants of urinary tract infection susceptibility. Curr Opin Infect Dis. 2015 Feb;28(1):88-96.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Lacerda Mariano L, Ingersoll MA. Bladder resident macrophages: Mucosal sentinels. Cell Immunol. 2018 Aug;330:136-141.\u00a0<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Song J, Abraham SN. TLR-mediated immune responses in the urinary tract. Curr Opin Microbiol. 2008 Feb;11(1):66-73.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Becknell B, Ching C, Spencer JD. The Responses of the Ribonuclease A Superfamily to Urinary Tract Infection. Front Immunol. 2019 Nov 29;10:2786.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Steigedal M, Marstad A, Haug M, Dam\u00e5s JK, Strong RK, et al. Lipocalin 2 imparts selective pressure on bacterial growth in the bladder and is elevated in women with urinary tract infection. J Immunol. 2014 Dec 15;193(12):6081-9.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Ueda N, Kondo M, Takezawa K, Kiuchi H, Sekii Y, et al. Bladder urothelium converts bacterial lipopolysaccharide information into neural signaling via an ATP-mediated pathway to enhance the micturition reflex for rapid defense. Sci Rep. 2020 Dec 3;10(1):21167.\u00a0<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Hayes BW, Abraham SN. Innate Immune Responses to Bladder Infection. Microbiol Spectr. 2016 Dec;4(6):10.1128\/microbiolspec.UTI-0024-2016.\u00a0<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">O\u2019Brien VP, Hannan TJ, Schaeffer AJ, Hultgren SJ. Are you experienced? Understanding bladder innate immunity in the context of recurrent urinary tract infection. Curr Opin Infect Dis. 2015 Feb;28(1):97-105.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Huang J, Fu L, Huang J, Zhao J, Zhang X, et al. Group 3 Innate Lymphoid Cells Protect the Host from the Uropathogenic Escherichia coli Infection in the Bladder. Adv Sci (Weinh). 2022 Feb;9(6):e2103303.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Wu J, Abraham SN. The Roles of T cells in Bladder Pathologies. 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Int J Med Microbiol. 2017 Dec;307(8):497-507.\u00a0<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Ambite I, Butler D, Wan MLY, Rosenblad T, Tran TH, Chao SM, Svanborg C. Molecular determinants of disease severity in urinary tract infection. Nat Rev Urol. 2021 Aug;18(8):468-486.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Ziegler T, Jacobsohn N, F\u00fcnfst\u00fcck R. Correlation between blood group phenotype and virulence properties of Escherichia coli in patients with chronic urinary tract infection. Int J Antimicrob Agents. 2004 Sep;24 Suppl 1:S70-5.<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Sulaiman KA, Al Qahtani N, Al Muqrin M, Al Dossari M, Al Wabel A, et al. The correlation between non-O blood group type and recurrent catheter-associated urinary tract infections in critically ill patients: A retrospective study. J Int Med Res. 2022 Jul;50(7):3000605221108082.\u00a0<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Albracht CD, Hreha TN, Hunstad DA. Sex effects in pyelonephritis. Pediatr Nephrol. 2021 Mar;36(3):507-515.\u00a0<\/p>\n<p><!-- \/wp:paragraph --><!-- wp:paragraph {\"style\":{\"elements\":{\"link\":{\"color\":{\"text\":\"var:preset|color|black\"}}}},\"textColor\":\"black\",\"fontSize\":\"small\"} --><\/p>\n<p class=\"has-black-color has-text-color has-link-color has-small-font-size\">Wnorowska U, Piktel E, Deptu\u0142a P, Wollny T, Kr\u00f3l G, et al. Ceragenin CSA-13 displays high antibacterial efficiency in a mouse model of urinary tract infection. Sci Rep. 2022 Nov 10;12(1):19164.<\/p>\t\t\t\t\t\t\t\t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t<\/div>","protected":false},"excerpt":{"rendered":"<p>La pared intestinal est\u00e1 formada por varias capas de tejidos y c\u00e9lulas\u00a0 especializadas cuya disposici\u00f3n en forma de peque\u00f1os pliegues llamados\u00a0 vellosidades intestinales le permite facilitar<span class=\"excerpt-hellip\"> [\u2026]<\/span><\/p>\n","protected":false},"author":1,"featured_media":1524,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[11],"tags":[],"class_list":["post-1459","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-salud-urologica"],"yoast_head":"<!-- This site is optimized with the Yoast SEO Premium plugin v24.7 (Yoast SEO v27.4) - https:\/\/yoast.com\/product\/yoast-seo-premium-wordpress\/ -->\n<title>Comprender la estructura del\u00a0 intestino | Urolistic<\/title>\n<meta name=\"description\" content=\"Redescubre el bienestar con Urolistic, el proyecto de la Dra. 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